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Controversial Cancer Stem Cells Offer New Direction For Treatment
In a review in Science, a University of Rochester Medical Center researcher sorts out the controversy and promise around a dangerous subtype of cancer cells, known as cancer stem cells, which seem capable of resisting many modern treatments.
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Molecule Helps Breast Cancer Cells To Survive In The Bone Marrow
Patients who survive an initial diagnosis of breast cancer often succumb to the disease years later when the cancer shows up in a different part of the body. Now, scientists have identified key signals that support the long term survival of breast cancer cells after they have spread to the bone marrow. The research, published by Cell Press in the July issue of the journal Cancer Cell, may lead to development of treatment strategies that decrease the likelihood of breast cancer recurrence in the bone and other organs. Drugshop to buy zoloft online and other pills.
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Proposed House Amendment Would Impact Needle Exchange Programs In Washington, D.C.
A proposed amendment to Washington, D.C."s federal appropriation for 2010 "would prohibit the city from using federal funds to distribute needles for the "injection of illegal drugs ņ€¦ within 1,000 feet of a public or private day care center, elementary school, vocational school, secondary school, college, junior college, university, public swimming pool, park, playground, video arcade or youth center,"" the Washington Post reports. Local HIV/AIDS advocates "are concerned that [the] proposed amendment ņ€¦ would drastically reduce public funding for needle exchange programs and take away a weapon in the fight against HIV and AIDS," according to the Post. A companion bill in the Senate does not contain language prohibiting the use of federal dollars for needle exchange programs. "Del. Eleanor Holmes Norton (D) urged her colleagues to fight the amendment which is before the full House this afternoon," the article states. The district has provided $700,000 in the past year to four non-profit organizations for needle exchange programs (Fears, 7/16).
Public Health

Maternally Inherited Neurological Disorder In Golden Retriever Dogs Is Caused By A Mutation In Mitochondrial DNA

Sensory ataxic neuropathy (SAN) is a recently identified neurological disorder in Golden Retriever dogs with onset during puppyhood. Affected dogs move in an uncoordinated manner and have sensory deficits. Researchers from the Swedish University of Agricultural Sciences, Uppsala University and the Karolinska Institutet have now revealed that SAN is caused by a mutation in mitochondrial DNA. The study is published May 29 in the open-access journal PLoS Genetics. The researchers were able to trace back all affected offspring on the maternal side, over more than 10 generations, to a female that lived during the 1970s. This implied a maternal inheritance, which was confirmed by the identification of a one base pair deletion in the mitochondrial tRNA-Tyr gene. Further analyses revealed that the mutation leads to mitochondrial dysfunction, which in turn causes a progressive loss of neurons. The researchers showed that about five percent of the current Swedish Golden Retriever population carries the mutation causing SAN. The identification of the mutation now allows genetic screening tests to identify carriers and prevent the mutation being transmitted to further generations. "This is a good example of how a close collaboration between clinicians and geneticists led to a rapid detection of a harmful mutation that can now be eliminated from this dog population to reduce suffering and disease," said co-author Karin Hultin Jē¤derlund. The study also provides a new animal model for similar mitochondrial disorders in humans, said co-author Izabella Baranowska, and could potentially be used for testing therapeutic approaches. CITATION: "Sensory Ataxic Neuropathy in Golden Retriever Dogs Is Caused by a Deletion in the Mitochondrial tRNATyr Gene." Baranowska I, Jē¤derlund KH, Nennesmo I, Holmqvist E, Heidrich N, et al. (2009) PLoS Genet 5(5): e1000499. doi:10.1371/journal.pgen.1000499 Plos Genetics


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